Vitamin D and AIDS link strengthening

NAM posted an interesting article on a paper today concerning the benefits of vitamin D in HIV patients. Given that my book supports HIV scepticism, I am going to comment on it from an alternate lens.

“Vitamin D insufficiency is associated with poorer CD4 cell recovery among women who start HIV treatment late… After taking into account factors such as viral load, the authors found a significant relationship between low levels of vitamin D and impaired immune reconstitution.”

i.e.  the outcome of a vitamin D system problem is the same as that of HIV infection.

“”Our study found that vitamin D insufficiency is associated with late CD4 recovery after HAART [highly active antiretroviral therapy] initiation,” comment the investigators.

HAART is toxic. Unarguably beneficial intermittently, and in low doses, but it’s still toxic. So CD4 depression isn’t surprising.

Vitamin D has an important role in overall health. Deficient levels have been linked to immune dysfunction, cardiovascular disease, impaired control of infections and depression.”


In the study, as you can read from the article, over a 24 month period, CD4 (severe depletion of which is also defined as AIDS in absence of HIV; so HIV is not an essential cause of AIDS even if it may be one) rose in tandem with vitamin D levels.

“Most of the women (60%) were African American and 89% had vitamin D insufficiency prior to starting HIV therapy.”

Ignoring even poverty this explains why Africa is hardest hit.

“Those with vitamin D insufficiency were more likely to aged over 38 years (94 vs 85%) and overweight or obese (98 vs 82%) than women with adequate levels of the vitamin.”

The likelihood of HIV infection increases with age (indeed seniors are shown to be getting it; late sex lives?), and yes, even obesity is associated with HIV infection.

“Follow-up at twelve months after the initiation of treatment showed that an undetectable viral load was similarly associated with a CD4 cell gain of 100 cells/mm3 or more at this time point (OR = 7.68; 95% CI, 3.46-17.03).”

So, again. Is the problem an infection (any infection), or the fact that the immune system is down in the first place?

“There may be biological mechanisms that explain the effect of vitamin D insufficiency on late CD4 cell recovery after HAART initiation,” comment the investigators. “Vitamin D insufficiency could be related to production of naïve T cells.”

HIV is said to kills T-cells but the mechanism is not known. This is like having a man in a room with a dead body. Did Man A kill Man B or is it just a co-incidence?
In this case we have to ask, does HIV kill T-cells or is the problem that they’re not being produced? i.e. Man A shot himself.

On World AIDS Day, consider not supporting a failing paradigm.


Be wary of National HIV Testing Week – in support of AIDS ‘denialism’

23-30 November is National HIV Testing Week, and for the first time it will be observed in England courtesy of the Terrence Higgins Trust.

The aim of this week is for consistent risk groups to come and get tested; that is gay men and Africans. However, given the fact that HIV-positivity doesn’t always progress to AIDS even without medication, that it doesn’t behave like a true infection by having remained within a cohort for 30 years, and that there are other holes in the HIV/AIDS hypothesis, I am asking such people (and even drug addicts) to consider refraining from testing. While I do not doubt that chronic HIV-positivity can indeed signify a health problem, questions remain as to whether HIV really does cause AIDS; and resultantly the medication given can equally do inappropriate harm as well as good.
My book features an extensive chapter on what the myriad causes of AIDS may be, plus a summation of HIV/AIDS hypothesis refutations. The role I adopt is that of HIV sceptic; I am not an AIDS denialist. In fact, most AIDS denialists are simply HIV sceptics.

December 1 is World AIDS Day, and perhaps you might consider some subterfuge: buy a strip of red ribbon, fashion a cut into a loop and wear it upside down. Most might think you’ve erred in pinning it; someone, though, might question what you’ve done and this can initiate argument.

Book follow-up: Improved bone density

This morning I received a verbal result for my latest bone mineral density scan (the reason for this procedure and previous results being outlined in my book), and I’m pleased to say that a lower dose of vitamin D3 (a little over 5000 IU daily) than previously did not result in a BMD decrease. In fact I actually gained a little more density even though I have never presented cause for concern. I don’t have exact figures as I wasn’t given a printout, and at this point I’m not even bothered with requesting them.

I required quite a lot of vitamin D when I first began my repletion journey but now 4-5000 IU daily is probably all I’ll ever need. That will sustain the inner fortress that I built!

To celebrate, for one day only, the Kindle edition of my book will be free to download from midnight tomorrow (17 Nov. 2012). The book doesn’t cost much anyway and is worth the admission fee, so if you miss out, help out an independent author and help yourself in the process.

London vitamin D conference in Feb 2013

It’s been a few years, to my knowledge, since the last local vitamin D event, but the Royal Society for Public Health have one arranged for 6 February 2013, featuring a host of British speakers.

Early bookers (before 14 December) can get a 10% discount. The price is still steep though, but any Londoners might like to let a medical professional know about it.

Read a PDF of the event here.

Support for Cannell’s theory of autism

Dr. John Cannell of the Vitamin D Council believes that vitamin D deficiency may be a contributor to autism. Here’s an item today that backs up his theory, albeit saying that more study is needed.

“…a twofold increased risk was seen for the more severe subtype of infantile autism (adjusted HR 2.3, 95% CI 1 to 5.3) following maternal influenza infection, according to Hjördis Ósk Atladóttir, MD, PhD, of the University of Aarhus in Denmark, and colleagues.”

If a mother has influenza her vitamin D levels will be significantly challenged to fight the infection, and if she is quite deficient to begin with, the amount of vitamin D she passes on to her foetus will also be compromised. Vitamin D appears crucial to brain development, as outlined in my book, but influenza on top for a growing life may have a negative effect as well.

“There also was a threefold increase in risk for infantile autism when mothers reported having had a febrile episode lasting for a week or more (aHR 3.2, 95% CI 1.8 to 5.6), the researchers reported online in Pediatrics.”

If a mother is feverish this is a sign that her immune system is quite compromised. Not a surprising scenario when you’re developing life in you and are fighting the flu. And are vitamin D deficient.

“Other than influenza, none of the infections were significantly associated with infantile autism, with aHRs ranging from 0.9 (95% CI 0.3 to 1.5) for genital herpes to 1.2 (95% CI 0.9 to 1.7) for cystitis
…There also was a small elevation in risk of the two [autism and spectrum] disorders with maternal use of antibiotics.”

Influenza and antibiotic usage probably provide the greatest hit to the immune system from the selection. The latter, certainly, temporarily makes the immune system redundant and does its role by proxy.

Instead of just pinning blame on post-natal interventions for autism we should really be looking earlier.

“Our results do not suggest that mild infections, febrile episodes, or use of antibiotics during pregnancy are strong risk factors for [autism spectrum disorder] and infantile autism,” Atladóttir and colleagues stated.

Maybe they indeed aren’t if a mother is at least vitamin D sufficient, as opposed to optimal. That is to say, they’re not a real threat with basic or great armour.